NR161 Mod 4 terms

high blood pressure

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Practice questions for this set

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alpha 1 antagonist- block alpha 2 receptors found in smooth muscle
alpha 2 antagonist- block alpha 2 receptors found in CNS and tissues

1

alpha antagonist

2

beta antagonist

3

muscarinic antagonist

4

alpha agonist

Terms in this set (23)

Hypertension

high blood pressure

essential hypertension

High blood pressure with no verifiable physical cause, which makes up the overwhelming majority of high blood pressure cases.

orthostatic hypertension

sudden increase in blood pressure when a person stands up

secondary hypertension

high blood pressure caused by the effects of another disease

Hypotension

low blood pressure

Adrenergic Receptors

receptor sites for the sympathetic neurotransmitters norepinephrine and epinephrine

agonists

drugs which mimic the activity of neurotransmitters

Angina

chest pain

Antagonists (drugs)

These drugs block the actions of neurotransmitters

antihypertensive therapy

Medications used to treat hypertension

adrengeric blocking medications

beta blockers ! block the effects of epinephrine and norepinephrine -> decreasing BP and HR

alpha antagonist

alpha 1 antagonist- block alpha 2 receptors found in smooth muscle
alpha 2 antagonist- block alpha 2 receptors found in CNS and tissues

beta 1 blocker

affects the heart

beta 2 blocker

affects the lungs

Cardiac Output

heart rate x stroke volume

Centrally acting adrenergic medications

Drugs that modify the function of the sympathetic nervous system in the brain by stimulating alpha2 receptors. Alpha2 receptors are inhibitory in nature and thus have a reverse sympathetic effect and cause decreased blood pressure and HR

Dysrhythmias

abnormal or irregular heartbeat

first-dose phenomenon

sudden drop in BP due to the intake of first dose of a new medication; can result in collapsing

intrinsic sympathomimetic activity

beta-blockers' ability to slightly activate the receptors they are trying to block -> blocks stress of some hormones, while stimulating intrinsic effects-> less pronounced HR

nonselective

drug that affects multiple receptors of the body, with no specific target

selective

drug targets specific receptors-> less likely to have a lot of side effects

sympatholytics

mechanisms that reduce the amount of norepinephrine released / inhibit the effects of sympathetic NS

vasodilation

Dilation of blood vessels -> increasing blood flow

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