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Lost Knowledge: Obesity
Without the will, there's still a way.
Oct 11, 2025
∙ Paid
Author’s note: A lot of difficult debate and review went into the decision to publish this article. By popular demand, with over 90% of respondents voting for publication, here it is. This is a rare kind of dangerous information; while everything here is, more or less, available elsewhere, piecemeal, and spread over dozens of sources of variable veracity, few so comprehensively cover both the risks and mitigations to be had, alongside these extraordinary actions. I hope that the compilation of this knowledge here will outweigh the risk of misuse. Please learn wisely, talk to your doctor, share as may be needed, and above all, be safe.
With great power comes great responsibility.
In every category that exists, everything from metallurgy, to education, to therapeutics, there’s a best-in-class; a gold standard, that one single approach that stands indisputably head-and-shoulders over the rest of the heap. When it comes to fighting ssRNA viral infections, at all phases of disease, ivermectin shines; for fly-with-a-bazooka immune suppression, it’s tacrolimus; for tensile strength at high temperature, it’s tungsten; once upon a time, before they were infected by communism, Harvard would have been best-in-class for education. I try to confine my work and my writings, on whichever subject they may be, to only the stand-out bests, and when it comes to weight loss, there’s two of them, depending on how you’re coming at it. The first, of course, is diet and exercise; no big secret there. If one expends more energy than they consume, and manages to keep their body out of starvation mode and actually burning those calories, those pounds of fat will melt off pretty quickly. Modern foods tend to pack enormous amounts of calories into relatively small portions; there’s a very good reason that the poorest among us also tend to be the heaviest. While RFK’s soda ban for food stamp recipients is a good starting point, when calorie-dense, processed junk food ends up cheaper than home-cooked, healthy meals, the problem runs a lot deeper than just soda.
However, there is a second route (and no, it’s not Ozempic.) The baseline principle is the same; with more energy out than in, fat gets burned. However, that common-sense approach has its limits. Obesity often comes with its own, self-reinforcing set of challenges. Even when the morbidly obese - among which we now count fully ten percent of Americans, with forty percent merely obese - finally do decide to take real steps toward fixing their health, they frequently find they are no longer able to do so, or only minimally so. Carrying all that weight around causes injuries, in and of itself; from back problems, to respiratory distress, or overloaded hips and joints creaking and cracking, the morbidly obese often find themselves in a catch-22, trapped in a prison of their own consumption, unable to do the right thing. Younger people are, of course, less likely to find themselves there, and generally, with the willpower to push through, can fight their way through it; but especially as aging takes its toll, that can cease to be the case.
A lot of knowledge was lost in the early 20th century. As the Rockefellers promulgated and cemented their profit-driven vision of medicine throughout the 1910s, 20s and 30s, establishing the labyrinthine system of medicine and the multi-trillion dollar sick-care industry that blights us today, new problems had to be created, and old ones brought back, to be facetiously “solved” again through the regulatory and educational morass that Dr. Fauci played like a fiddle to exact his genocide against the world, and the US particularly, throughout the pandemic. Among those pieces of knowledge, banned in the late 1930s, due to the (very real) risks of overdose - as exist for any drug, most notably and recently Tylenol - was the extraordinary, gold-standard weight loss effects of 2,4-Dinitrophenol.
With Ozempic, generally held by the world as the current gold standard, four pounds of weight loss, per month, is hailed as a miracle. While in context of what one would expect from good old diet and exercise, it’s nothing to sneeze at - presuming you don’t mind the “safe and effective” side effects - it’s the kiddie pool compared to what’s possible. Four pounds, depending on one’s starting point, might be roughly what you’d expect to see in one single day of treatment with 2,4-DNP. The drug stands alone in a category all its own when it comes to burning fat. Of course, like any drug, it comes with pitfalls and side effects, some of them quite dangerous. Most of the therapeutics that I’ve talked about, to date, have had extremely minimal risks. Ivermectin, for treating all manner of ssRNA viral infections, has one of the best safety records of any drug in history, magnified further when compared to the mortality of some such infections; fenbendazole and menbendazole, with notable but manageable risks to liver health, are nevertheless far safer than what may, often, be guaranteed mortality from all manner of cancers; ascorbic acid, the inimitable Vitamin C, has a very well known safety profile.
I’ll tell you right now: one of these is not like the others. 2,4-DNP carries very real risks, when misused or overdosed, up to and including blindness, or a death that is certainly no less unpleasant than a Tylenol overdose. The therapeutic window, which defines the useful range of dosage of a drug between having no effect, at too low a dose, or having deleterious effects, in overdose, is substantially narrower than most drugs available today; and its unique mechanisms, and even the typical psychology of a person using it, also contribute to magnify that risk with anything but the most disciplined, carefully planned and compliant treatment regime.
Nevertheless, when used correctly and cautiously, the drug can be safe, effective, and truly orders of magnitude more effective than anything else available today. Let’s get into what it is, how it works, and why, when nothing else will do, 2,4-Dinitrophenol is the undisputed king of weight loss. I’m surely going to catch some flak for even talking about it, and many of the criticisms that I anticipate will, generally speaking, be well grounded; this is a high voltage, high risk, high reward therapeutic for obesity and weight loss. If you find yourself considering it, I implore you to treat it with the utmost respect and care that it demands, and as with any other therapeutic protocol, to seek medical advice and supervision before and during its use; this article does not substitute for personalized, professional medical advice from a practitioner that knows your circumstances.
A profound example of the suppression of “dangerous” knowledge surfaced quite recently; although we’ve been talking about the likelihood of cancer, from the mRNA shots, for the better part of five years now, it has remained a minority position. Those who can read the science, or prefer independent journalism over mainstream or establishment publications, knew full well what was coming, whilst the majority were told it was safe. Meanwhile, in an expose recently published by Pfizer whistleblower, Brook Jackson, it was revealed that Sen. Ron Johnson, and a few of the so-called leaders of the largely now-captured Covid resistance, also knew all about it, but were explicitly instructed to withhold or slow-walk that information, because it might be scary - and they complied. With friends like these…
While this is still dangerous, for completely different reasons, the principle stands: knowledge is power, and I would rather empower you to make your own, informed choices. The “experts” of the day didn’t think you could be trusted with these truths.
Prove them wrong, and be safe.
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What is 2,4-Dinitrophenol?
2,4-Dinitrophenol is in a rare class of drugs known as ionophores. Best known in modernity as a chemical pesticide and dye, this unique molecule acts on the inner mitochondrial membrane, the boundary layer between the mitochondrial matrix and the intermembrane space, and disrupts the delicate proton balance across that boundary. All extant life today - everything, from a tomato plant, to a tardigrade, to every single cell in your body and that of literally every living organism today, bar none - depends upon the adenosine triphosphate molecule, ATP, to generate and transport energy within and between cells. Red blood cells carry oxygen; ATP carries energy.
If ATP is the gas can, then the mitochondria are the refineries; in one of the best examples of the machine-like nature of our microbiology, the ATP molecule is synthesized, at that boundary, by the aptly named ATP synthase protein. It is an engine in every sense of the word: fuel, the raw materials to produce ATP are injected, it physically spins and rearranges those materials, and energetic ATP molecules are released to power all the functions critical to life, along with a little heat. This process happens millions of times a second in every single metabolizing cell. It is not the only means of producing ATP, but as one of several energy-producing mechanisms, which we evolved over time to preferentially engage, our metabolism heavily depends upon it. The others are, broadly, substrate phosphorylation and anaerobic fermentation, which utilize completely different pathways in different circumstances. Here, we are converting fats to ketones, and then to energy; those pathways will be familiar to those who have investigated or undertaken the keto, as short for ketosis, dietary strategies. That’s also a valid approach, and one that has yielded excellent results for many people; it’s also a lot less risky and, of course, a lot less effective, especially in light of the operation of the homeostatic weight setpoint, which unlike many other methods, 2,4-DNP does appear to act upon, leading to weight loss that actually persists.
The operation of ATP synthase depends on an ionic imbalance between the interior and the exterior of the mitochondrial membrane. It is, almost (but not quite) literally, an electrical circuit; the voltage differential between the inside and the outside of the cell transmits chemical energy across the ATP synthase engine, which consumes that electric current, spins that electric motor, drains the charge outside of the cell, and as with any electrical device, puts off a little waste heat. Proton pumps act as the chargers; consuming energy themselves, albeit less than the synthase protein produces, they maintain that voltage differential by continuously pumping protons, hydrogen ions, which cross the membrane to power that engine, back out of the mitochondrial matrix and into the intermembrane space again, where the cycle repeats. It runs frequently, but not constantly: enzymes within the cell, AMP-activated protein kinase (AMPK) specifically, tell the mitochondria when more ATP energy is required, and the mitochondria activates those proteins until that energy demand is met, generating heat and oxidative waste products. The process is called oxidative phosphorylation: that chemo-electric fuel is literally burned, or oxidized, to add a phosphate group to the precursor ADP, adenosine diphosphate. If ATP is the gas can when it’s full, ADP is the very same can, but when it’s empty; lacking that extra molecule of phosphate, it needs to be refilled, by ATP synthase, for reuse.
DNP takes a sledgehammer to that delicately balanced circuit. In the ordinary course of events, the potential energy in that mitochondrial battery is carefully discharged across ATP synthase, powering that engine. DNP is, effectively, a short circuit. It diffuses across that membrane, and provides a path through which those protons can simply cross straight over the membrane. In doing so, they do no useful work, depriving ATP synthase of the energy it needs to operate; if the intermembrane space is the positive terminal, the mitochondrial matrix the negative, and ATP synthase is the controlled, well-oiled engine sitting between them, DNP is a thick strand of copper directly between the terminals, short-circuiting that battery as fast as it can carry those protons across the boundary. If you’re at all familiar with what happens in a short circuit…
…it applies just as equally here. That potential energy still does work, in the thermodynamic sense, and still directly proportional to the amount of energy being discharged across the membrane; but rather than useful work, just as in a short circuit, that energy is exclusively dissipated as heat, and quite a bit of it, at that.
That short-circuit has numerous effects. The first, most obvious one, is heating: if you bridge a short circuit, you’re going to make a very hot wire. Doing so also depletes the energy that’s available for ATP synthase to do its job, and the proton pumps have to work overtime to recharge that battery; for the same amount of energy that’s expended across the membrane, far less ATP is actually produced. There is, of course, only so much fuel for those reactions that’s actually available; even as ATP becomes much more difficult to produce, the cell’s energy needs don’t magically disappear. The first and most easily accessible source of energy is sugars, glycogen specifically. They are stored primarily, by volume, in the muscles; glycogen is to ATP as crude oil is to gas. Muscles, the primary site of bioavailable glycogen storage, have two metrics by which they can be judged, being their density, and their volume. Muscles can either grow larger, and in doing so, store more glycogen, which gives them enhanced endurance over time; or, they can grow denser, which increases raw strength. (Or both.) That dichotomy is why the lean martial artist, whilst perhaps not possessed of the stamina of a completely ripped bodybuilder, can nevertheless hit twice as hard, and easily win an arm wrestle against him. This is also one of the fundamental differences between men and women; the fairer sex, generally, develops a less dense musculature.
As DNP does its work, shuttling protons from the intermembrane space back into the mitochondrial matrix to waste enormous amounts of energy, the raw fuel requirement of that cell skyrockets. The first place from which it will draw that energy is the glycogen stores. This is, obviously, completely non-conducive to weight loss, and as we will see, one of the primary causes of overdose and death in inexperienced or impatient users of this drug. Just as in ketosis, for any effective weight loss to occur, the body must first deplete its most readily available energy source, being that sugary glycogen, before any fat reserves will be tapped. That process can typically take several days, being directly proportional to the amount of glycogen immediately available. Throughout that period, absolutely no noticeable changes or effects will occur.
The complete lack of any instant result is infamous for frustrating impatient users who, despite having perhaps started at a safe dose, then go on to take much more than a safe dose, hoping to see some effect, and only belatedly, when almost nothing can then be done, realize their tragic error in having exceeded that narrow therapeutic window, as it begins to take effect.
Feeling The Burn
Once that period has elapsed, though, it’s go time. Glycogen stores are depleted, and with every cell in the body screaming for energy, an unbridled assault on the body’s fat reserves commences, by the same method, albeit greatly accelerated, as the keto diet. The first symptom to be noticed, aside from the rise in body temperature, is the extreme tiredness. Glycogen depletion is probably more familiar to you as being tired. If you just ran a marathon, your muscles are going to be tired. Congratulations: one is now, sitting and sweating in an air-conditioned room, because they don’t have the energy to do anything else, running that marathon, every minute of every day, until the body finally, eventually metabolizes and disposes of that DNP.
DNP is lipophilic; it binds to fatty acids. This is, of course, a core function of how it operates; cellular membranes are made of lipids, and that lipophilicity is what enables it to diffuse across the cell membranes until it reaches its targets on the mitochondria. It also attaches to lipocytes, those cells of fat that you’re trying to burn, and is only slowly released into the bloodstream to find a target, and to be metabolized and excreted, over the course of approximately three days. This has an extremely dangerous effect on the undisciplined, inexperienced, or impatient user, and again, mirrors pretty closely the deadly course of a Tylenol overdose; if you overdose today, you’re not going to know, or feel any different, for at least a few days’ time, and once those three days have passed, the active dose in your system is going to rise, proportionally to dose, and continue to rise for another day or two.
This is how death by overdose occurs. The DNP that was taken a few days ago is only now, finally, becoming active; even if one realizes their error, and completely ceases taking it, it’s far too late to make a difference. This might also be a good time to mention that there is no antidote. Death, by malignant hyperthermia and multiple organ failure, is the standard outcome of an acute overdose; typical cooling measures, such as ice baths, dantrolene, or even intravenous cooling, have very low success rates, regardless of how quickly the overdose is caught or treated. It is, by all accounts, an extremely painful, violent, and distressing way to die; unlike an ordinary heat stroke, that extreme, excess heat is being generated from the inside, by every single affected cell, and it’s simply impossible to reach and cool them all fast enough. In a few, very mild cases, there have been some successes, but as a rule, the vast majority of overdoses will result in a painful death, and that should be the expected result of diverging, for any reason, from a safely, conservatively calculated dosing regime. Overdosing on DNP will be the last mistake you ever make.
Let me repeat that:
OVERDOSING ON 2,4-DINITROPHENOL WILL BE THE LAST MISTAKE YOU EVER MAKE.
So, you didn’t overdose - now what?
Congratulations! Now you have a week, maybe two at the outer limits, to enjoy your total exhaustion: as you lay still in bed, or your favorite comfy chair, sweating until the sheets are soaked through (and dyed yellow, but we’ll get to that one), you are spending every minute of every day running a marathon, the intensity of which is proportional to the dose of DNP that you took and maintained. Avoiding an overdose is just the beginning; now we need to take a closer look at exactly what’s happening as all that fat and energy is being burned.
You may be familiar with lactic acid. As you exert yourself in the course of ordinary physical activity, lactic acid builds up in your muscles; when it starts to hurt, that might be your sign that it’s time to take a break; or you can, with great willpower, push through the burn, dream the impossible dream, and finish those 20 push-ups. However, you’re in luck - you don’t need a drop of that willpower now! Here’s to pharmaceutical miracles! No matter what you do, you’re going to be feeling that burn, and it’s just going to keep getting worse until you’re done. So, we need to find another way to deal with that.
Aside from overdose, the next biggest risk of DNP comes from the overproduction of reactive oxygen species: you might better know them as free radicals. They are produced in the normal course of exertion, and in that normal course of exertion, they are produced in a reasonable number that your body can naturally handle; while some people like to take antioxidants as a general health supplement, and they can in fact help to further mitigate the damage they do and the visible aging that they cause, they generally aren’t strictly necessary to live a healthy, normal life.
However, running a 24-7 marathon is not something that normal people normally do.
With inhuman levels of energy (and fat) being burned - dose-dependently, up to or over twice that of an ordinary resting metabolism - inhuman levels of free reactive oxygen species, AKA free radicals, are now being produced in every cell in the body. If you aren’t familiar with the meaning of the term “reactive oxygen species,” put it this way - hydrogen peroxide, also known as a bleach, is one of the more common reactive oxygen species that we see and use every day, and that is exactly what you’re producing - and the chemical results are about what you might imagine. This over-production of ROS is the primary mechanism by which the second-most common side effect of DNP occurs: blindness.
This can and will happen, if not continuously mitigated, with an otherwise safe dose.
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However, the news on this front is much better than a simple overdose, and brings us to the second vital element of what is known, at least in the bodybuilding community, as a DNP cycle: frequent, heavy doses of unsugared electrolytes and antioxidants.
Reactive oxygen species are just that; unstable, highly reactive oxygen radicals, looking for another chemical with which they can react. Oxygen is everywhere, and reacts with pretty much everything. If you’ve ever seen a rusty car sitting in a yard, you can see the results: oxygen reacts with iron, iron oxide ensues, and the car is slowly rusted away and destroyed. This also happens to be one of the primary mechanisms of premature aging, and the primary reason that various antioxidant supplements are so popular. However, as you run that race, you burn months of energy for every single day that goes by; free radicals are being produced at inhuman rates, far beyond what the body would normally see, and far in excess the body’s natural capacity to metabolize them. Unchecked, they will cause extensive tissue damage. While most cells in the body can, to some extent, handle some oxidative stress, the eyes are much less forgiving: especially in women, an abundance of free radicals will oxidize - also known as burning - the lipids and proteins of the lens and the cornea, leading to cataract formation. The inevitable, extensive sweating also causes electrolyte depletion. Most ‘sports drinks’ are also heavily sugared, which as we’ll cover later, is counterindicated; suitable sports supplements, sans sugars, do exist.
Now, that better news that I mentioned is twofold. Firstly, although it’s nothing that anybody ever wants to have to go through, that particular form of blindness is treatable, with surgical success rates up to 98%; cataract surgery, in which the clouded lens is replaced, is almost as commonplace as plastic surgery now, and in the modern world, so long as you’ve got insurance or the money to cover it, is quite effective. As that good news goes, though, that’s decidedly the less good.
The better news is that it is avoidable. The favorite antioxidant, all over, is … drum roll please, for the inimitable … Vitamin C! Ascorbic acid is one of the strongest antioxidants known to man, and is broadly systemically bioavailable. Unlike my various other protocols, which call for a specific form of it, being pure, powdered, oral ascorbic acid, generally for reasons of rapid absorption and immunomodulatory function, the precise form in which it’s taken here does not matter at all. Quantity over quality.
Glutathione also serves a similar purpose; as another antioxidant, and in another interesting Tylenol parallel, it too will be consumed by the extensive ROS production, and will benefit from supplementation. While direct oral glutathione is poorly absorbed, the prodrug N-acetylcysteine, or NAC, which should be well known to any who investigated useful Covid therapeutics, enhances its availability.
So long as there’s a hefty and continuous stream of antioxidants and electrolytes being made available to the body - and given the quantity that will be passing through the stomach, continuously throughout, a gentler form of Vitamin C, such as sodium ascorbate, is generally preferred - those free radicals will have all the antioxidants in the world with which to harmlessly react, and electrolyte balance can be maintained. Thereafter, instead of a death by a thousand cuts at the hands of a lake of hydrogen peroxide, that reaction forms, after a short chain of further reactions, dehydroascorbic acid (as well as some oxygen and water); that harmless, metabolized end-form of ascorbic acid, which is excreted primarily through the urine (of which one will be producing plenty), almost completely mitigates the presently superhuman metabolism:
And there you have it! With this knowledge, and your doctor’s advice and consent -please be aware that I am not a medical doctor, this is not medical advice, and I cannot be responsible for your use or misuse of this dangerous, educational information - one can run a 24 hour, seven to fourteen day, non-stop marathon, from the comfort of their favorite piece of newly yellow, soaking wet furniture, and by the end of it, find themselves up to forty plus pounds lighter.
Yep. That’s not a typo. Believe me though, as I speak from experience, one will feel every bit as drained and exhausted as though you had been out there on that track. It’s difficult to really say that it replaces the need for willpower, per-se; whilst having commenced a cycle, given the pharmacokinetics I have described for you above, one will have little choice but to see it through, long past the point at which you’re tired and would like to go home now, please, you will certainly remember the extraordinary exertion it entails the next time you consider taking it. Having completed one lower-dose, test cycle, I found that it took every bit as much willpower to commence the next one as it would to have gone out on that track. The results, however, are truly in a league of their own. Once upon a time, at 5’7”, I was 242 pounds, with a respectable beer and cheeseburger belly; after a single week’s cycle, and subject to some further nuance I will cover below, I finished that month at a rather less-rounded 198 pounds. That was almost a decade ago now, and even without much in the way of lifestyle change, I never put that weight back on, either; the homeostatic weight setpoint, the consternation of crash dieters since the beginning of time, does appear to reset to wheresoever one lands. I should also note that that was with a relatively high dose; I arguably unwisely skated the edge, but having done the math, I was confident in where it landed, and as it turns out - I’m still here! - and a decade later, I am still at about that same weight.
The fine print…
That is, in sum, the best and the worst of it. Of course, there is a great deal of care and consideration that must go into avoiding those negative outcomes. We’ve covered the essentials; I hope that I have struck a good balance between communicating the unparalleled efficacy of 2,4-Dinitrophenol, warning you of the very real risks, and describing - heretofore, in broad strokes - what it takes to avoid them. There are also some unique quirks to its effects, and the progressive timeline of a cycle, that can be, at first, quite confusing and counter-intuitive, that I will go into.
The legal status of 2,4-DNP across the US, and the world, is quite interesting. Broadly, it has been treated by regulatory regimes not as a drug, but as an agricultural or industrial chemical; for that reason, it sits in a particularly unique grey area. It has been banned as a supplement or drug since 1938; it remains illegal to sell it for medical purposes. However, it is not illegal to buy it, or - usually, with some exceptions, definitely check your local laws - to possess it in small quantities; in larger quantities, far more than are needed for any potential pharmaceutical use, some regulatory regimes treat it as an explosive or a poison. Of course, now that I’ve published this, that could quite conceivably change any time. For those reasons, reputable, lawful suppliers tend to be international. Ukraine, particularly, has been known for producing and selling it in forms very definitely not intended for human consumption; purity and measurements can vary materially, and with such an unforgivingly narrow therapeutic window, and a near-complete absence of regulatory oversight in “manufacturing,” buyer beware when it comes to trusting the label.
Given that, for the last hundred years, formal scientific research, study or consideration of 2,4-DNP has largely been limited to case studies of overdoses and deaths, the following knowledge of its actual use, derived from the original studies over 100 years ago, has largely been obtained and refined by the trial, and occasional horrific error, of the bodybuilding and weightlifting communities, and of course, my own personal studies and experience. It’s a relatively thin knowledge base, especially in light of the overflowing wealth of information available on some of the other subjects I have explored, but in my opinion, it is quite sufficient for cautious use.
The most typical application of 2,4-DNP, beyond angsty, anorexic teenage girls that think they’re fat, is bodybuilders looking to rapidly cut their fat before a contest; whether to better define their muscular figure, or to sneak in under the line of a lower weight class, the extreme rapidity of its action makes it invaluable to such people looking for an edge. Medicinal use, for the treatment of obesity, tends to be much rarer, but not unheard of. Interestingly, the drug has also served as a Russian secret weapon in their deployment of General Winter; in several wars, Russian conscripts, fighting through the howling European winters with deeply inadequate clothing, were issued low doses of DNP to elevate their metabolisms, keeping them warm as they trudged through the sub-freezing steppes. It was, reportedly, highly effective.
The dye aspect of it is profound: it is an extremely potent, systemic yellow dye. Urine will take on a characteristic, particularly deep yellow hue, distinct from, and somewhat masking of, dehydration; the ocular sclera, and even the skin, can similarly be affected, which also has the unfortunate effect of mimicking, to some extent, the appearance of jaundice, albeit, except in cases of overdose where it would be the least of one’s problems, not exhibiting any particular hepatotoxicity. Surfaces, objects and fabrics, if exposed, will be permanently stained upon exposure; as mentioned above, sweat will be an ever present companion throughout a cycle, and similarly carries the dye; while I found that darker colors tended to be unaffected, or at least much less notably so, whites will certainly cease to be, so with white furnishings, clothes or bedsheets, if one would like them to remain so, it is a consideration to be made.
The process has a built-in time limit: cycles tend to be limited to a week, or up to two at absolute maximum. Beyond that, a temporary tolerance develops; in response to the glaring inefficiencies it produces, more ATP synthase is deployed by cells, the drug is cleared faster, different metabolic pathways take over, and the useful effect is sharply limited.
2,4-DNP is not metabolized by any of the usual enzymatic drug clearance pathways, such as CYP3A4 or CYP450; it is largely excreted intact, in urine, and of the fraction that is processed, that occurs via glucuronidation and sulfation in the liver, with no known (further) toxic metabolites.
The Don’ts
Heavy exercise during a cycle is generally frowned upon and counterproductive. You wanted to lose serious weight without physical exercise or willpower; you got it, so “enjoy” it. Consumption of carbs or sugars - bread, candy, soda - is also a particularly bad idea; beyond the obvious effect of replenishing glycogen stores, which defeats the entire purpose and methodology, people have reported - myself among them - a flash of intense heat upon consumption, above and beyond what’s already occurring, as those sugars are rapidly metabolized. On top of everything else, you’re still going on a brief keto diet anyway, too. Have some meat and cheese.
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